[Expression of NF-kappaB/IkappaB signal pathway in renal tissues of hepatitis B virus-associated nephritis]

Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2006 May;22(3):327-9.
[Article in Chinese]

Abstract

Aim: To investigate the expression of nuclear transcription factor-kappa B (NF-kappaB)/IkappaB in renal tissues of hepatitis B virus-associated nephritis (HBVGN) and to study its role in the pathogenesis of HBVGN.

Methods: The expression of NF-kappaB p65 and IkappaB-alpha in renal tissues was examined in biopsy specimens from 42 HBVGN patients, 20 patients with primary membranous nephropathy and 5 normal subjects by immunohistochemical staining.

Results: The NF-kappaB p65 expression in glomerular and tubular of HBVGN tissues was notably higher than that in normal control tissues (P<0.05) and in primary membranous nephropathy tissues (P<0.01). The IkappaB-alpha expression in glomerular and tubular of HBVGN tissues was lower than that in primary membranous nephropathy tissues (P<0.05).

Conclusion: There might be protein degradation of IkappaB-alpha and nuclear translocation of NF-kappaB in renal tissues of HBVGN, which suggests that NF-kappaB/IkappaB signal pathway may play an important role in the pathogenesis of HBVGN.

MeSH terms

  • Adolescent
  • Adult
  • Female
  • Gene Expression
  • Glomerular Basement Membrane / metabolism*
  • Hepatitis B / complications
  • Hepatitis B virus / pathogenicity
  • Humans
  • I-kappa B Proteins / metabolism*
  • Kidney Tubules / metabolism
  • Male
  • NF-KappaB Inhibitor alpha
  • NF-kappa B / metabolism*
  • Nephritis / etiology
  • Nephritis / metabolism*
  • Nephritis / virology
  • Signal Transduction / physiology*
  • Transcription Factor RelA / metabolism*
  • Young Adult

Substances

  • I-kappa B Proteins
  • NF-kappa B
  • NFKBIA protein, human
  • Transcription Factor RelA
  • NF-KappaB Inhibitor alpha