Regulation by the cAMP cascade of oxygen free radical balance in mammalian cells

Antioxid Redox Signal. 2006 Mar-Apr;8(3-4):495-502. doi: 10.1089/ars.2006.8.495.

Abstract

A study is presented of the effect of the cAMP cascade on oxygen metabolism in mammalian cell cultures. Serum-starvation of the cell cultures resulted in depression of the forward NADH-ubiquinone oxidoreductase activity of complex I, decreased content of glutathione, and enhancement of the cellular level of H2O2. Depressed transcription of cytosolic Cu/Zn-SOD 1, mitochondrial glutathione peroxidase and catalase was also observed. Activation of the cAMP cascade reversed the depression of the activity of complex I and the accumulation of H2O2. The effect of cAMP involved the cAMP-dependent protein kinase.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Catalase / chemistry
  • Cyclic AMP / metabolism*
  • Cyclic AMP-Dependent Protein Kinases / chemistry
  • Cytosol / enzymology
  • Fibroblasts / metabolism
  • Free Radicals*
  • Glutathione Peroxidase / chemistry
  • Humans
  • Hydrogen Peroxide / pharmacology
  • Mice
  • Mice, Inbred BALB C
  • NIH 3T3 Cells
  • Oxygen / chemistry*
  • Reactive Oxygen Species
  • Superoxide Dismutase / chemistry

Substances

  • Free Radicals
  • Reactive Oxygen Species
  • Hydrogen Peroxide
  • Cyclic AMP
  • Catalase
  • Glutathione Peroxidase
  • Superoxide Dismutase
  • Cyclic AMP-Dependent Protein Kinases
  • Oxygen