[Expression of major histocompatibility complex antigens and intercellular adhesion molecule-1 (ICAM-1) on renal cell cancer. De novo expression and modulation by cytokines on renal cell cancer cell lines]

Nihon Hinyokika Gakkai Zasshi. 1991 Feb;82(2):232-8. doi: 10.5980/jpnjurol1989.82.232.
[Article in Japanese]

Abstract

Major histocompatibility complex (MHC) antigens and intercellular adhesion molecule-1 (ICAM-1) play important roles in immune response. In order to investigate the association between renal cell cancer (RCC) and host's immune system, expression of MHC antigens and ICAM-1 was examined on RCC. Immunohistochemical analysis revealed a positive correlation between the expression of MHC antigens and ICAM-1. In general, tumor with higher degree of mononuclear cell infiltration expressed MHC antigens and ICAM-1 more frequently and intensely. Among cytokines which were reported to be potent inducers of ICAM-1 on malignant melanoma cell lines, interferon (IFN)-gamma and tumor necrosis factor (TNF)-alpha augmented the expression of ICAM-1 on ACHN cells whereas ICAM-1 and class I antigens on KRC/Y cells. IFN-alpha enhanced MHC class I antigens but not ICAM-1. Class II antigen expression of both cell lines was augmented by only IFN-gamma. These results suggest that cytokines which could be produced by tumor-infiltrating mononuclear cells, especially IFN-gamma and TNF-alpha, might modulate expression of MHC antigens and ICAM-1, and influence host immune response against RCC.

Publication types

  • English Abstract

MeSH terms

  • Adult
  • Aged
  • Carcinoma, Renal Cell / immunology*
  • Cell Adhesion Molecules / analysis*
  • Female
  • Histocompatibility Antigens Class I / analysis*
  • Histocompatibility Antigens Class II / analysis*
  • Humans
  • Immunoenzyme Techniques
  • Intercellular Adhesion Molecule-1
  • Interferon-gamma / biosynthesis
  • Interferon-gamma / physiology*
  • Kidney Neoplasms / immunology*
  • Leukocytes, Mononuclear / immunology
  • Leukocytes, Mononuclear / metabolism
  • Male
  • Middle Aged
  • Tumor Cells, Cultured
  • Tumor Necrosis Factor-alpha / biosynthesis
  • Tumor Necrosis Factor-alpha / physiology*

Substances

  • Cell Adhesion Molecules
  • Histocompatibility Antigens Class I
  • Histocompatibility Antigens Class II
  • Tumor Necrosis Factor-alpha
  • Intercellular Adhesion Molecule-1
  • Interferon-gamma