Nasal polyposis in Peutz-Jeghers syndrome: a distinct histopathological and molecular genetic entity

J Clin Pathol. 2007 Apr;60(4):392-6. doi: 10.1136/jcp.2005.036418. Epub 2006 Jun 14.

Abstract

Background: Peutz-Jeghers syndrome (PJS) is an autosomal dominant hamartomatous polyposis syndrome of the gastrointestinal tract, caused by a germline STK11/LKB1 mutation. Nasal polyposis was described in the original report by Peutz. Recently, a molecular-genetic association between nasal polyposis and PJS has been reported.

Objective: To further explore the occurrence and pathogenesis of PJS-related nasal polyposis.

Methods: 51 patients with PJS, 84 unaffected family members and 36 spouses from 18 families with PJS were questioned for the presence of nasal polyposis. 12 PJS-related nasal polyps, 1 carcinoma of the nasal cavity and 28 sporadic nasal polyps were analysed for loss of (wild type) STK11/LKB1, eosinophilia, squamous metaplasia, dysplasia and expression of cyclo-oxygenase 2 and p53.

Results: Nasal polyps occurred in 8 of 51 patients with PJS, and were not reported by non-affected family members (p<0.001). Germline STK11/LKB1 mutations were identified in all patients with PJS and nasal polyposis. Loss of heterozygosity was found in four of eight PJS-related nasal polyps, but not in sporadic nasal polyps (p = 0.002). PJS-related nasal polyps showed less eosinophilia than sporadic nasal polyps (p<0.001). Expression of cyclo-oxygenase 2 was found in 11 of 12 PJS-related nasal polyps and 19 of 28 sporadic nasal polyps (p>0.05). Overexpression of p53 was not found.

Conclusions: Nasal polyposis occurs in a significant number of Dutch patients with PJS, one of whom developed a carcinoma in the nasal cavity. The loss of heterozygosity, and the absence of eosinophilia suggest a distinct pathogenesis compared with sporadic nasal polyposis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • AMP-Activated Protein Kinase Kinases
  • Adolescent
  • Child
  • Cyclooxygenase 2 / metabolism
  • Eosinophilia / pathology
  • Female
  • Germ-Line Mutation
  • Humans
  • Loss of Heterozygosity
  • Male
  • Middle Aged
  • Nasal Polyps / genetics*
  • Nasal Polyps / metabolism
  • Nasal Polyps / pathology
  • Peutz-Jeghers Syndrome / genetics*
  • Peutz-Jeghers Syndrome / metabolism
  • Peutz-Jeghers Syndrome / pathology
  • Protein Serine-Threonine Kinases / genetics
  • Tumor Suppressor Protein p53 / metabolism

Substances

  • Tumor Suppressor Protein p53
  • Cyclooxygenase 2
  • Protein Serine-Threonine Kinases
  • STK11 protein, human
  • AMP-Activated Protein Kinase Kinases