Phagocytosis of N-acetyl-D-glucosamine particles, a Th1 adjuvant, by RAW 264.7 cells results in MAPK activation and TNF-alpha, but not IL-10, production

Cell Immunol. 2006 Feb;239(2):103-12. doi: 10.1016/j.cellimm.2006.04.003. Epub 2006 Jun 16.

Abstract

A practical and highly effective Th1 adjuvant should induce Th1 cytokines (IL-12, IL-18, and TNF-alpha) but not the Th2 cytokine IL-10, an inhibitor of Th1 responses. In this study, phagocytosis of N-acetyl-d-glucosamine polymer (chitin) particles by RAW 264.7 cells, a murine macrophage-like cell line, resulted in phosphorylation of MAPK (p38, Erk 1/2, and JNK), and production of relatively high levels of TNF-alpha and COX-2 with increased PGE(2) release. Similar results were observed in response to oligonucleotides with CpG motifs, mycobacterial components and endotoxin. However, these bacterial components also induced a large amount of IL-10. Chitin particles, in contrast, failed to induce detectable levels of IL-10, although the production of high levels of PGE(2) and TNF-alpha and the activation of MAPK's are potentially positive signals for IL-10 production. Thus, our results indicate that chitin particles act as a unique Th1 adjuvant for macrophages without inducing increased production of IL-10.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Acetylglucosamine / metabolism*
  • Adjuvants, Immunologic / metabolism*
  • Adjuvants, Immunologic / physiology
  • Animals
  • Cell Line
  • Chitin / metabolism
  • Enzyme Activation / immunology
  • Female
  • Interleukin-10* / biosynthesis
  • Macrophages / enzymology
  • Macrophages / metabolism*
  • Mice
  • Mice, Inbred C57BL
  • Mitogen-Activated Protein Kinases / metabolism*
  • Particle Size
  • Phagocytosis / immunology*
  • Th1 Cells / immunology
  • Th1 Cells / metabolism*
  • Tumor Necrosis Factor-alpha / biosynthesis*

Substances

  • Adjuvants, Immunologic
  • Tumor Necrosis Factor-alpha
  • Interleukin-10
  • Chitin
  • Mitogen-Activated Protein Kinases
  • Acetylglucosamine