A direct mechanistic link between growth control and a tumor cell immune function: increased interleukin-8 secretion accounts for elimination of Oct-1 antisense transformants from scid mice

Anticancer Res. 2006 May-Jun;26(3A):1733-8.

Abstract

Background: Tumorigenesis involves the aberrant function of proteins that regulate growth control, including Oct-1. Oct-1 is a DNA binding transcription factor that activates genes that encode proteins required for S-phase and cell growth. For example, Oct-1 activates the histone H2B promoter and the promoters for the snRNPs. Oct-1 also represses certain promoters, including promoters of immune function genes, such as the IL-8 and the HLA-DRA genes.

Materials, methods and results: Oct-1 antisense transformants were determined to have reduced growth rates and other characteristics of growth control. Also, Oct-1 antisense transformants endured for a shorter time in scid mice, being attributable to the increased expression of IL-8 by the Oct-1 antisense transformants.

Conclusion: These results may help resolve the conundrum of why growth control de-regulation alone is not enough for tumorigenicity. The results also support the conclusion that the molecular mechanisms of growth control de-regulation and tumor cell immune functions are directly linked.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Cell Growth Processes / genetics
  • Cell Growth Processes / immunology
  • DNA, Antisense / genetics
  • Humans
  • Interleukin-8 / biosynthesis
  • Interleukin-8 / genetics
  • Interleukin-8 / immunology
  • Interleukin-8 / metabolism*
  • Mice
  • Mice, SCID
  • Neoplasm Transplantation
  • Octamer Transcription Factor-1 / genetics*
  • Transformation, Genetic
  • Transplantation, Heterologous
  • Urinary Bladder Neoplasms / genetics
  • Urinary Bladder Neoplasms / immunology*
  • Urinary Bladder Neoplasms / metabolism
  • Urinary Bladder Neoplasms / pathology*

Substances

  • DNA, Antisense
  • Interleukin-8
  • Octamer Transcription Factor-1