Chinese hamster ovary cell-derived recombinant human acid alpha-glucosidase in infantile-onset Pompe disease

J Pediatr. 2006 Jul;149(1):89-97. doi: 10.1016/j.jpeds.2006.02.035.

Abstract

Objective: To conduct an open-label, multinational, multicenter study examining the safety and efficacy of recombinant human acid alpha-glucosidase (rhGAA) in treatment of infantile-onset Pompe disease.

Study design: We enrolled 8 infant patients who had Pompe disease with GAA activity <1% of normal, cardiomyopathy, and hypotonia. In the 52-week initial phase, rhGAA was infused intravenously at 10 mg/kg weekly; an extension phase continued survivors' treatment with 10 to 20 mg/kg of rhGAA weekly or 20 mg/kg every 2 weeks for as long as 153 weeks. Safety measurements included adverse events, laboratory tests, and anti-rhGAA antibody titers. Efficacy evaluations included survival, ventilator use, echocardiograms, growth, and motor and cognitive function.

Result: After 52 weeks of treatment, 6 of 8 patients were alive, and 5 patients were free of invasive ventilator support. Clinical improvements included ameliorated cardiomyopathy and improved growth and cognition. Five patients acquired new motor milestones; 3 patients walked independently. Four patients died after the initial study phase; the median age at death or treatment withdrawal for all patients was 21.7 months, significantly later than expected for patients who were not treated. Treatment was safe and well tolerated; no death was drug-related.

Conclusion: rhGAA improved ventilator-free survival, cardiomyopathy, growth, and motor function in patients with infantile-onset Pompe disease compared with outcomes expected for patients without treatment.

Publication types

  • Clinical Trial, Phase II
  • Multicenter Study
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Body Height / drug effects
  • Body Weight / drug effects
  • Cardiomyopathy, Hypertrophic / drug therapy
  • Cardiomyopathy, Hypertrophic / etiology
  • Child Development
  • Europe / epidemiology
  • Female
  • Glycogen / metabolism
  • Glycogen Storage Disease Type II / complications
  • Glycogen Storage Disease Type II / drug therapy*
  • Glycogen Storage Disease Type II / mortality*
  • Hearing Disorders / etiology
  • Humans
  • Infant
  • Infant, Newborn
  • Infusions, Intravenous
  • Male
  • Muscle Hypotonia / drug therapy
  • Muscle Hypotonia / etiology
  • Muscle, Skeletal / metabolism
  • Muscle, Skeletal / pathology
  • Respiration, Artificial
  • Treatment Outcome
  • United States / epidemiology
  • alpha-Glucosidases / metabolism
  • alpha-Glucosidases / therapeutic use*

Substances

  • Glycogen
  • GAA protein, human
  • alpha-Glucosidases