Induction of murine interleukin-1 beta expression by water-soluble components from Hericium erinaceum

Acta Pharmacol Sin. 2006 Aug;27(8):1058-64. doi: 10.1111/j.1745-7254.2006.00321.x.

Abstract

Aim: To investigate the inductive effect of water extract from Hericium erinaceum (WEHE) on interleukin-1beta (IL-1beta) expression.

Methods: A murine macrophage cell-line, RAW 264.7 was stimulated with 1 to 10 mg/L WEHE and inductions of IL-1beta protein and its steady state mRNA were examined using a bioassay, Western blotting, and reverse transcription-polymerase chain reaction (RT-PCR) analysis. The inductive effect of WEHE on IL-1beta gene expression was further investigated by a chloramphenicol acetyltransferase (CAT) reporter gene assay using a transient transfection with pIL-1(870 bp)-CAT where the expression of the CAT gene was regulated by a IL-1beta promoter. An electrophoretic mobility shift assay (EMSA) was also performed to examine transcription factors, nuclear factor-kappa B (NF-kappaB), activator protein 1 (AP-1), nuclear factor interleukin-6 (NF-IL6), and cAMP response element (CRE)/activating transcription factor (ATF).

Results: WEHE induced IL-1beta production in both its mRNA and protein expression in a dose-dependent manner. The inductive effect of WEHE on IL-1beta gene expression was due to the augmentation of the IL-1beta transcription. Furthermore, EMSA showed that WEHE markedly increased the binding activities of NF-kappaB, and to a lesser extent, those of AP-1 and NF-IL6 to their cognate DNA recognition sites, whereas CRE/ATF binding remained constant, all of which are known to be involved in the regulation of IL-1beta gene expression.

Conclusion: WEHE induces IL-1kappa expression in macrophages at a transcriptional level by enhancing the activation of transcription factors, NF-kappaB, NF-IL6, and AP-1.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Agaricales* / chemistry
  • Animals
  • Biological Products / administration & dosage
  • Biological Products / isolation & purification
  • Biological Products / pharmacology*
  • CCAAT-Enhancer-Binding Protein-beta / metabolism
  • Cell Line
  • Dose-Response Relationship, Drug
  • Interleukin-1beta / biosynthesis*
  • Interleukin-1beta / genetics
  • Macrophage Activation / drug effects
  • Macrophages / cytology
  • Macrophages / metabolism*
  • Mice
  • Mice, Inbred C3H
  • NF-kappa B / metabolism
  • RNA, Messenger / biosynthesis
  • RNA, Messenger / genetics
  • Transcription Factor AP-1 / metabolism
  • beta-Glucans / administration & dosage
  • beta-Glucans / isolation & purification
  • beta-Glucans / pharmacology*

Substances

  • Biological Products
  • CCAAT-Enhancer-Binding Protein-beta
  • Interleukin-1beta
  • NF-kappa B
  • RNA, Messenger
  • Transcription Factor AP-1
  • beta-Glucans