Abstract
There is now considerable evidence from both experimental and clinical studies that immune and inflammatory processes can contribute to the onset of stroke and the neurologic and psychologic outcomes. Several specific therapeutic targets have been identified that may significantly improve the devastating impact of stroke.
MeSH terms
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Animals
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Brain / immunology*
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Brain Damage, Chronic / immunology*
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Brain Damage, Chronic / psychology
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Cell Death / immunology
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Cerebral Hemorrhage / immunology*
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Cerebral Hemorrhage / psychology
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Cerebral Infarction / immunology*
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Cerebral Infarction / psychology
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Chemokines / blood
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Cytokines / immunology
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Encephalomyelitis / immunology
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Encephalomyelitis / psychology
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Free Radicals / metabolism
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Humans
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Microglia / immunology
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Neurons / immunology
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Psychoneuroimmunology
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Sick Role
Substances
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Chemokines
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Cytokines
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Free Radicals