mTOR-independent translational control of the extrinsic cell death pathway by RalA

Mol Cell Biol. 2006 Oct;26(20):7345-57. doi: 10.1128/MCB.00126-06. Epub 2006 Aug 7.

Abstract

Oncogenic potential is associated with translational regulation, and the prevailing view is that oncogenes use mTOR-dependent pathways to up-regulate the synthesis of proteins critical for transformation. In this study, we show that RalA, a key mediator of Ras transformation, is also linked to the translational machinery. At least part of this linkage, however, is independent of mTOR and acts through RalBP1 to suppress cdc42-mediated activation of S6 kinase and the translation of the antiapoptotic protein FLIP(S). This action, rather than contributing to transformation, opens a latent tumor-suppressive mechanism that can be activated by tumor necrosis factor-related apoptosis-inducing ligand. These results show that the translational machinery is linked to tumor suppression as well as cell-proliferative pathways and that the reestablishment of cell death pathways by activation of the Ral oncogenic program provides a means for selective therapeutic targeting of Ral-driven malignancies.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • ATP-Binding Cassette Transporters / genetics
  • ATP-Binding Cassette Transporters / metabolism
  • Animals
  • Apoptosis Regulatory Proteins / metabolism
  • Astrocytes / cytology
  • Astrocytes / metabolism
  • CASP8 and FADD-Like Apoptosis Regulating Protein
  • Cell Death
  • Cell Line
  • Gene Expression Regulation
  • Humans
  • Intracellular Signaling Peptides and Proteins / metabolism
  • Membrane Glycoproteins / metabolism
  • Mice
  • Phosphorylation
  • Protein Binding
  • Protein Biosynthesis*
  • Protein Kinases / genetics
  • Protein Kinases / metabolism*
  • Ribosomal Protein S6 Kinases / genetics
  • Ribosomal Protein S6 Kinases / metabolism
  • Signal Transduction
  • TNF-Related Apoptosis-Inducing Ligand
  • TOR Serine-Threonine Kinases
  • Tumor Necrosis Factor-alpha / metabolism
  • cdc42 GTP-Binding Protein / genetics
  • cdc42 GTP-Binding Protein / metabolism
  • ral GTP-Binding Proteins / genetics
  • ral GTP-Binding Proteins / metabolism*

Substances

  • ATP-Binding Cassette Transporters
  • Apoptosis Regulatory Proteins
  • CASP8 and FADD-Like Apoptosis Regulating Protein
  • CFLAR protein, human
  • Cflar protein, mouse
  • Intracellular Signaling Peptides and Proteins
  • Membrane Glycoproteins
  • TNF-Related Apoptosis-Inducing Ligand
  • TNFSF10 protein, human
  • Tnfsf10 protein, mouse
  • Tumor Necrosis Factor-alpha
  • Protein Kinases
  • MTOR protein, human
  • mTOR protein, mouse
  • Ribosomal Protein S6 Kinases
  • TOR Serine-Threonine Kinases
  • cdc42 GTP-Binding Protein
  • ral GTP-Binding Proteins