Clinical features and gene analysis in Korean patients with early-onset Parkinson disease

Arch Neurol. 2006 Aug;63(8):1170-4. doi: 10.1001/archneur.63.8.1170.

Abstract

Background: Systematic analysis of clinical features and gene mutations has not been performed in Korean patients with early-onset Parkinson disease (PD).

Objective: To investigate the clinical characteristics and genetic background of Korean patients with early-onset PD.

Design: Clinical and genetic study.

Setting: University hospital.

Patients: Ninety-four patients with early-onset PD (mean +/- SD age at onset, 39.8 +/- 7.3 years) of 1100 patients with PD.

Interventions: Analysis of clinical characteristics and mutation analysis of the parkin and PTEN-induced kinase (PINK1) genes by direct sequencing and gene-dosage analysis using the multiplex ligation-dependent probe amplification technique.

Main outcome measures: The correlation between age at onset and clinical characteristics and the clinical features of patients with onset before age 30 years vs patients with onset after age 30 years.

Results: Because age at onset was younger, levodopa-induced dyskinesia and off-dystonia were more frequently observed (P=.008). Patients affected before age 30 years showed more frequent levodopa-induced dyskinesia and off-dystonia (P=.002). We identified 3 patients (5%) with parkin gene mutations but none with the PINK1 mutation.

Conclusions: Earlier onset of levodopa-induced dyskinesia and off-dystonia were characteristic features of early-onset PD, especially before an onset age of 30 years. However, parkin gene mutations were less frequent in these patients than in Japanese groups reported elsewhere.

Publication types

  • Clinical Trial
  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Age of Onset
  • Dyskinesia, Drug-Induced / epidemiology
  • Dyskinesia, Drug-Induced / genetics
  • Female
  • Humans
  • Korea / epidemiology
  • Levodopa / adverse effects
  • Levodopa / therapeutic use
  • Male
  • Middle Aged
  • Mutation*
  • Parkinson Disease / drug therapy
  • Parkinson Disease / epidemiology*
  • Parkinson Disease / genetics*
  • Protein Kinases / genetics
  • Ubiquitin-Protein Ligases / genetics

Substances

  • Levodopa
  • Ubiquitin-Protein Ligases
  • parkin protein
  • Protein Kinases
  • PTEN-induced putative kinase