Genetic variability in CHMP2B and frontotemporal dementia

Neurodegener Dis. 2006;3(3):129-33. doi: 10.1159/000094771.

Abstract

A nonsense/protein chain-terminating mutation in the CHMP2B gene has recently been reported as a cause of frontotemporal dementia (FTD) in the single large family known to show linkage to chromosome 3. Screening for mutations in this gene in a large series of FTD families and individual patients led to the identification of a protein-truncating mutation in 2 unaffected members of an Afrikaner family with FTD, but not in their affected relatives. The putative pathogenicity of CHMP2B mutations for dementia is discussed.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Amino Acid Sequence
  • Dementia / genetics*
  • Endosomal Sorting Complexes Required for Transport
  • Female
  • Genetic Predisposition to Disease*
  • Humans
  • Male
  • Middle Aged
  • Molecular Sequence Data
  • Mutation
  • Nerve Tissue Proteins / genetics*
  • Pedigree
  • Polymerase Chain Reaction

Substances

  • CHMP2B protein, human
  • Endosomal Sorting Complexes Required for Transport
  • Nerve Tissue Proteins