Increased resistance to beta-lactams in a Klebsiella pneumoniae strain: role of a deletion downstream of the Pribnow box in the blaSHV-1 promoter

Int J Antimicrob Agents. 2006 Oct;28(4):308-12. doi: 10.1016/j.ijantimicag.2006.07.004. Epub 2006 Sep 7.

Abstract

Two hundred and five isolates of Klebsiella pneumoniae were collected from Nantes University Hospital in 2002. A new 30bp deletion was detected downstream of the -10 box of the SHV-1 promoter in a clinical K. pneumoniae isolate with a high amoxicillin/clavulanic acid minimum inhibitory concentration. Reverse transcription polymerase chain reaction revealed increased transcription of bla(SHV-1) mRNA. All conjugation mating assays failed. This new promoter was cloned upstream of the cat gene of the reporter plasmid pKK232-8 and compared with previously described bla(SHV-1) promoters. The deletion induced a 15-fold increase in promoter strength compared with the usual weak promoter. This study reports a new genetic event that increases bla(SHV-1) chromosomal gene expression, which may be of clinical relevance when associated with porin deficiency.

MeSH terms

  • DNA, Bacterial / analysis
  • Drug Resistance, Bacterial / genetics
  • Gene Deletion*
  • Gene Expression Regulation, Bacterial / drug effects
  • Klebsiella Infections
  • Klebsiella pneumoniae / drug effects*
  • Klebsiella pneumoniae / enzymology
  • Klebsiella pneumoniae / genetics
  • Klebsiella pneumoniae / metabolism
  • Microbial Sensitivity Tests
  • Plasmids / genetics
  • Promoter Regions, Genetic*
  • beta-Lactam Resistance / genetics*
  • beta-Lactamases / genetics
  • beta-Lactamases / metabolism*
  • beta-Lactams / pharmacology*

Substances

  • DNA, Bacterial
  • beta-Lactams
  • beta-lactamase PIT-2
  • beta-Lactamases