High-efficient lentiviral vector-mediated gene transfer into primary human NK cells

Exp Hematol. 2006 Oct;34(10):1344-52. doi: 10.1016/j.exphem.2006.06.001.

Abstract

Objective: The long-term transfection of genes into primary natural killer (NK) cells without disrupting normal cellular functions has been proven to be difficult with currently available gene-transfer methods. In this study, we establish a lentiviral vector-based technique for improved gene transfer into human NK cells in vitro and we report on high-efficient transduction of freshly isolated as well as cultured primary NK cells.

Methods: Freshly isolated or primary cultured human NK cells, as well as the human NK cell line YTS, were transduced with replication-incompetent human immunodeficiency virus (HIV)-based lentiviral vector bearing a GFP reporter gene or a gene of interest under the control of the elongation factor 1alpha (EF1alpha) promoter. Transduction efficiencies were monitored by flow cytometry.

Results: A long-term transgene expression was detected in up to 98% of YTS NK cells, whereas in freshly isolated or primary cultured NK cells exposed to interleukin (IL)-2 plus IL-12 upon infection, efficiency was in the range of 50% to 90%. Moreover, in freshly isolated quiescent NK cells a transfection efficiency of 18% to 20% was achieved without stimulation. Notably, no major phenotypic and functional modifications were observed in transduced cells with respect to control cells: the expression levels of activating receptors, CD69-antigen induction as well as cytotoxic function were unaffected.

Conclusion: Results of our study demonstrate that NK cells can be efficiently transduced by lentiviral vectors.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigens, CD / biosynthesis
  • Antigens, Differentiation, T-Lymphocyte / biosynthesis
  • Cells, Cultured
  • Genes, Reporter
  • Genetic Vectors*
  • Humans
  • Interleukin-12 / pharmacology
  • Killer Cells, Natural* / cytology
  • Killer Cells, Natural* / metabolism
  • Lectins, C-Type
  • Lentivirus*
  • Lymphocyte Activation / drug effects
  • Lymphocyte Activation / genetics
  • Peptide Elongation Factor 1 / genetics
  • Promoter Regions, Genetic / genetics
  • Time Factors
  • Transduction, Genetic* / methods

Substances

  • Antigens, CD
  • Antigens, Differentiation, T-Lymphocyte
  • CD69 antigen
  • Lectins, C-Type
  • Peptide Elongation Factor 1
  • Interleukin-12