G-CSF enhanced granulocyte production in a child with severe neutropenia in the setting of fatal virus-associated hemophagocytic syndrome

Am J Hematol. 1990 Dec;35(4):266-8. doi: 10.1002/ajh.2830350409.

Abstract

A 14-year-old boy with fatal varicella zoster virus-associated hemophagocytic syndrome (VAHS) was treated with recombinant human granulocyte colony-stimulating factor (G-CSF) based on the finding that the patient had severe neutropenia and possible bacterial superinfection. Support for the G-CSF therapy in VAHS is provided by the recent reports that G-CSF is relatively specific for the granulocyte lineage; it would not activate mature monocyte/macrophage/histiocyte lineage in VAHS, where the most striking morphologic feature is histiocytic hyperplasia with hemophagocytosis. He responded well to G-CSF with an elevation of neutrophil counts. There were no effects on other blood cells. The result indicates that G-CSF is useful to increase granulocyte production in severe neutropenia, even in the setting of fatal VAHS.

Publication types

  • Case Reports

MeSH terms

  • Adolescent
  • Autopsy
  • Granulocyte Colony-Stimulating Factor / therapeutic use*
  • Granulocytes / cytology*
  • Hematopoiesis / drug effects
  • Histiocytosis, Non-Langerhans-Cell / complications
  • Histiocytosis, Non-Langerhans-Cell / drug therapy*
  • Humans
  • Male
  • Neutropenia / complications*

Substances

  • Granulocyte Colony-Stimulating Factor