Efects of growth hormone and cadmium on the transcription regulation of two metallothionein isoforms

Mol Cell Endocrinol. 2007 Jan 15;263(1-2):29-37. doi: 10.1016/j.mce.2006.08.010. Epub 2006 Oct 5.

Abstract

The effect of growth hormone (GH) and cadmium (Cd) on metallothionein (MT) expression was investigated in hepatoma cells. In fish the constitutive isoform MT-B and the metal-responsive MT-A are expressed. Real-time RT-PCR revealed that: Cd up-regulates mostly MT-A, GH slightly induces MT-B and the GH/Cd combination induces synergistically both MTs. Perturbations in Ca2+ levels suppressed or reduced the Cd-induction of MTs and abolished the GH/Cd synergy. Similar results were obtained by inhibition of tyrosine kinases. Also the signaling molecules recruited by the GH receptor responded differently to GH and Cd, with ERKs showing a synergistic activation upon GH/Cd. The following conclusions can be drawn: (1) cytosolic Ca2+ is mainly involved in MT-A regulation; (2) both Ca2+ and tyrosine phosphorylation are essential for Cd-induction and GH/Cd synergy on MTs. The synergy could depend on interactions in different signaling pathways, leading to a differential recruitment of MTF-1 and AP-1 transcription factors.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cadmium / pharmacology*
  • Calcium / metabolism
  • Carcinoma, Hepatocellular / genetics*
  • Carcinoma, Hepatocellular / metabolism
  • DNA-Binding Proteins / metabolism
  • Drug Synergism
  • Gene Expression Regulation / drug effects*
  • Growth Hormone / pharmacology*
  • Liver Neoplasms / genetics
  • Liver Neoplasms / metabolism
  • Metallothionein / genetics*
  • Metallothionein / metabolism
  • Mitogen-Activated Protein Kinases / metabolism
  • Oncorhynchus mykiss / metabolism
  • Phosphorylation / drug effects
  • Promoter Regions, Genetic
  • Protein Binding
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • Receptors, Somatotropin / metabolism
  • Reverse Transcriptase Polymerase Chain Reaction
  • Transcription Factor AP-1 / metabolism
  • Transcription Factor MTF-1
  • Transcription Factors / metabolism
  • Transcription, Genetic
  • Tumor Cells, Cultured
  • Tyrosine / metabolism

Substances

  • DNA-Binding Proteins
  • RNA, Messenger
  • Receptors, Somatotropin
  • Transcription Factor AP-1
  • Transcription Factors
  • Cadmium
  • Tyrosine
  • Growth Hormone
  • Metallothionein
  • Mitogen-Activated Protein Kinases
  • Calcium