The effects of increasing serum calcitriol on energy and fat metabolism and gene expression

Obesity (Silver Spring). 2006 Oct;14(10):1739-46. doi: 10.1038/oby.2006.200.

Abstract

Objective: Evidence from a number of investigations indicates that calcium intake could be inversely related to body weight through alterations in the 1,25-OH(2)-D(3) metabolism. The objective of this study was to test whether energy and substrate metabolism and adipose tissue enzyme mRNA expression can be altered by changes in serum 1,25-OH(2)-D(3) through oral cholecalciferol supplementation in non-obese human subjects.

Research methods and procedures: An intervention study was used with a treatment period of 7 days. During this intervention, energy expenditure (EE) and substrate metabolism were measured using indirect calorimetry at t = 0, 1, 3, and 7 days, and blood samples were obtained at t = -1, 0, 1, 2, 3, 5 and 7 days. Fat biopsies were obtained at t = 0 and 7 days for determination of expression of genes involved in lipolytic and lipogenic pathways. Subjects from the general community were studied in an ambulatory setting at a university hospital. Ten healthy young men (age, 28 +/- 3 years; BMI, 25.5 +/- 0.5 kg/m(2)) were recruited by local announcement, and all completed the study. All subjects received 2000 IU cholecalciferol/d for 7 days, and they were instructed to consume a low-cholecalciferol, low-calcium diet. EE, fat oxidation, and adipose tissue enzyme mRNA were the main outcome measures.

Results: Despite a significant increase in serum 1,25-OH(2)-D(3) concentration at t = 5 and 7 days, no significant differences in substrate and energy metabolism nor mRNA concentrations of different lipid metabolism-related proteins were observed.

Discussion: Seven-day supplementation with 2000 IU cholecalciferol/d together with a decrease in dietary calcium intake does not affect EE or substrate metabolism nor gene expression of proteins related to fat metabolism, despite a significant increase in serum 1,25-OH(2)-D(3) concentration.

Publication types

  • Clinical Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adipose Tissue / drug effects*
  • Adipose Tissue / metabolism
  • Adult
  • Analysis of Variance
  • Blood Glucose / metabolism
  • Calcitriol / blood*
  • Calcium / blood
  • Cholecalciferol / administration & dosage
  • Cholecalciferol / blood
  • Cholecalciferol / pharmacology*
  • Dietary Supplements
  • Energy Metabolism / drug effects*
  • Fatty Acid Synthases / genetics
  • Fatty Acids, Nonesterified / blood
  • Gene Expression / drug effects*
  • Gene Expression / genetics
  • Glycerol / blood
  • Glycerolphosphate Dehydrogenase / genetics
  • Humans
  • Ion Channels / genetics
  • Male
  • Mitochondrial Proteins / genetics
  • PPAR gamma / genetics
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • Reverse Transcriptase Polymerase Chain Reaction
  • Sterol Esterase / genetics
  • Time Factors
  • Uncoupling Protein 2
  • Vitamins / administration & dosage
  • Vitamins / blood
  • Vitamins / pharmacology

Substances

  • Blood Glucose
  • Fatty Acids, Nonesterified
  • Ion Channels
  • Mitochondrial Proteins
  • PPAR gamma
  • RNA, Messenger
  • Uncoupling Protein 2
  • Vitamins
  • Cholecalciferol
  • Glycerolphosphate Dehydrogenase
  • Fatty Acid Synthases
  • Sterol Esterase
  • Calcitriol
  • Glycerol
  • Calcium