New potential ligand-receptor signaling loops in ovarian cancer identified in multiple gene expression studies

Cancer Res. 2006 Nov 15;66(22):10709-19. doi: 10.1158/0008-5472.CAN-06-1327. Epub 2006 Nov 6.

Abstract

Based on the hypothesis that gene products involved in the same biological process would be coupled at transcriptional level, a previous study analyzed the correlation of the gene expression patterns of ligand-receptor (L-R) pairs to discover potential autocrine/paracrine signaling loops in different cancers (Graeber and Eisenberg. Nat Genet 2001; 29:295). By refining the starting database, a list of 511 L-R pairs was compiled, combined to eight data sets from a single pathology, epithelial ovarian cancer, and examined as a proof-of-principle of the statistical and biological validity of the correlation of the L-R gene expression patterns in cancer. Analysis revealed a Bonferroni-corrected significant correlation of 105 L-R pairs in at least one data set and, by systematic analysis, identified 39 more frequently correlated L-R pairs, 7 of which were already biologically confirmed. In four data sets examined for an L-R correlation associated with patient survival time, 15 L-R pairs were significantly correlated in short surviving patients in two of the data sets. Immunohistochemical analysis of one of the newly identified correlated L-R pairs (i.e., EFNB3-EPHB4) revealed the correlated expression of ephrin-B3 and EphB4 proteins in 45 of 55 epithelial ovarian tumor samples (P < 0.0001). Together, these data not only support the validity of cross-comparison analysis of gene expression data because known and expected correlations were confirmed but also point to the promise of such analysis in identifying new L-R signaling loops in cancer.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Ephrin-B3 / biosynthesis
  • Ephrin-B3 / genetics
  • Ephrin-B3 / metabolism
  • Epithelial Cells / pathology
  • Female
  • Gene Expression Profiling
  • Humans
  • Immunohistochemistry
  • Ligands
  • Ovarian Neoplasms / genetics*
  • Ovarian Neoplasms / metabolism*
  • Receptor, EphB4 / biosynthesis
  • Receptor, EphB4 / genetics
  • Receptor, EphB4 / metabolism
  • Receptors, Cell Surface / biosynthesis
  • Receptors, Cell Surface / genetics
  • Receptors, Cell Surface / metabolism*
  • Signal Transduction

Substances

  • Ephrin-B3
  • Ligands
  • Receptors, Cell Surface
  • Receptor, EphB4