Multidrug resistance reversal by 3-formylchromones in human colon cancer and human mdr1 gene-transfected mouse lymphoma cells

In Vivo. 2006 Sep-Oct;20(5):645-9.

Abstract

Several new 3-formylchromone derivatives proved to be modifiers of multidrug resistance in mouse lymphoma cells and in human Colo320 colon cancer cells. There is apparently a structure-activity relationship between the antiproliferative multidrug resistance-reversing effect and the chemical structure of the 3-formylchromones. The total polar surface areas and the ground state dipole moments of the molecules are presumed to play a key role in the multidrug resistance-reversing effect. The log P values can provide an adequate explanation for the selective cytotoxicity against cancer cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Line, Tumor
  • Cell Proliferation
  • Chromones / chemistry
  • Chromones / pharmacology*
  • Colonic Neoplasms / drug therapy*
  • Drug Resistance, Multiple*
  • Drug Resistance, Neoplasm*
  • Genes, MDR*
  • Humans
  • Lymphoma, T-Cell / drug therapy*
  • Mice
  • Molecular Structure
  • Transfection

Substances

  • Chromones
  • formylchromone