Antigen-independent T cell activation mediated by a very late activation antigen-like extracellular matrix receptor

Eur J Immunol. 1991 Jun;21(6):1559-62. doi: 10.1002/eji.1830210634.

Abstract

Cell-cell and cell-extracellular matrix (ECM) binding mediated by integrin molecules has been implicated in lymphocyte migration and adhesion. We describe here that ECM binding triggers antigen-independent activation of T cell functions. Fibronectin and vitronectin, when coated on plates, not only acted synergistically on anti-CD3-induced serine esterase release in a murine cytotoxic T lymphocyte clone and interleukin 2 production in a murine helper T cell hybridoma but also could trigger these responses alone. All these stimulatory effects of ECM were abrogated by a monoclonal antibody which reacts with a unique very late activation antigen-like integrin and this monoclonal antibody, when coated on plates, exhibited a similar synergistic effect to that of ECM proteins. Therefore, ECM receptors expressed on activated T cells appear to play an important role in triggering T cell effector functions in localized tissues abundant in ECM proteins.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigens, Differentiation, T-Lymphocyte / physiology
  • CD2 Antigens
  • CD3 Complex
  • Extracellular Matrix Proteins / physiology*
  • Fibronectins / pharmacology
  • Glycoproteins / pharmacology
  • Humans
  • Lymphocyte Activation*
  • Receptors, Antigen, T-Cell / physiology
  • Receptors, Immunologic / physiology
  • T-Lymphocytes / immunology*
  • Vitronectin

Substances

  • Antigens, Differentiation, T-Lymphocyte
  • CD2 Antigens
  • CD3 Complex
  • Extracellular Matrix Proteins
  • Fibronectins
  • Glycoproteins
  • Receptors, Antigen, T-Cell
  • Receptors, Immunologic
  • Vitronectin