Silica-ceramic suppresses the expression of proinflammatory cytokines induced by lipopolysaccharide in macrophages

J Biomed Mater Res A. 2007 Mar 1;80(3):513-9. doi: 10.1002/jbm.a.31106.

Abstract

Bioactive materials have previously been used to coat implants. In a new development for bioactive materials, a silica-ceramic mixture was found to alleviate pain (Lee, Poster presented at the Ninth World Congress of Gynecological Endocrinology, Hongkong, 2001. Poster session (p47)). Here, we hypothesized that silica-ceramic can reduce the expression and activity of cyclooxygenase 2 (COX2) or cytokines associated with inflammation. The production of COX2 and proinflammatory cytokines was investigated by reverse transcriptase (RT)-PCR and ELISA assay in macrophages stimulated by lipopolysaccharide (LPS). Silica-ceramic had no effect of COX2 expression and prostaglandin production in macrophages. However, silica-ceramic suppressed the synthesis of cytokines involved in inflammation, in particular, the expression of IL-1beta and IL-6 was reduced at the transcriptional and translational levels. The involvement of NF-kappaB in the suppression of cytokines by silica-ceramic was examined by luciferase reporter assay. The NF-kappaB activity stimulated by LPS was inhibited by 20-60% with silica-ceramic compared with treatment with LPS alone. We suggest that inhibition of NF-kappaB activity by silica-ceramic might cause the attenuation of proinflammatory cytokine expression in macrophages. In conclusion, silica-ceramic could be an alternative approach to regulate the inflammation process.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Line
  • Ceramics / pharmacology*
  • Cyclooxygenase 2 / genetics
  • Cytokines / genetics*
  • Gene Expression Regulation / drug effects*
  • Inflammation / immunology
  • Interleukin-1beta / genetics
  • Interleukin-6 / genetics
  • Lipopolysaccharides / pharmacology
  • Macrophages / drug effects*
  • Mice
  • NF-kappa B / antagonists & inhibitors*
  • Silicon Dioxide / pharmacology*

Substances

  • Cytokines
  • Interleukin-1beta
  • Interleukin-6
  • Lipopolysaccharides
  • NF-kappa B
  • Silicon Dioxide
  • Cyclooxygenase 2