Centrally administered galanin inhibits osmotically stimulated arginine vasopressin release in conscious rats

Neurosci Lett. 1991 Jul 22;128(2):245-8. doi: 10.1016/0304-3940(91)90271-t.

Abstract

The effect of centrally administered galanin on arginine vasopressin (AVP) release was investigated in conscious rats. Intracerebroventricular injection of porcine galanin suppressed hypertonic saline-induced increase in plasma AVP in a dose-dependent manner (12.5-100 pmol/rat) at 10 min after the injection. Pretreatment with subcutaneous injection of naloxone (1 mg/100 g b.wt.) partially blocked the galanin-induced effect on plasma AVP. These results suggest that central galanin inhibits osmotically stimulated AVP release and endogenous opioids are, at least in part, involved in the mechanism.

MeSH terms

  • Animals
  • Arginine Vasopressin / metabolism*
  • Dose-Response Relationship, Drug
  • Galanin
  • Hypertonic Solutions / pharmacology
  • Injections, Intraventricular
  • Male
  • Naloxone / pharmacology
  • Osmolar Concentration
  • Peptides / administration & dosage
  • Peptides / pharmacology*
  • Rats
  • Rats, Inbred Strains

Substances

  • Hypertonic Solutions
  • Peptides
  • Arginine Vasopressin
  • Naloxone
  • Galanin