TP53 and TP53-related genes associated with protection from apoptosis in the radioadaptive response

Radiat Res. 2007 Jan;167(1):51-7. doi: 10.1667/RR0623.1.

Abstract

We investigated the effect of administering priming low-dose radiation prior to high-dose radiation on the level of apoptosis and on the expression of TP53 and TP53-related genes in mouse splenocytes. The percentage of apoptotic cells was significantly lower in TP53(+/+) mice receiving priming radiation 2 to 168 h before the high-dose irradiation, compared to TP53(+/+) mice exposed to 2 Gy alone. In contrast, TP53(+/-) mice exhibited a reduced level of apoptosis only when priming was performed for 2 or 4 h prior to the high-dose irradiation. In TP53(+/+) mice, primed mice had higher TP53 expression than mice exposed to 2 Gy. Phospho-TP53 (ser15/18) expression was the highest in mice exposed to 2 Gy and intermediate in primed mice. Expression of p21 (CDKN1A) was higher in primed mice compared with mice exposed to 2 Gy. MDM2 expression remained at a high level in all mice receiving 2 Gy. Elevated phospho-ATM expression was observed only in mice exposed to 2 Gy. We conclude that TP53 plays a critical role in the radioadaptive response and that TP53 and TP53-related genes might protect cells from apoptosis through activation of the intracellular repair system.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis*
  • Cyclin-Dependent Kinase Inhibitor p21 / biosynthesis*
  • DNA Repair
  • Dose-Response Relationship, Radiation
  • Gene Expression Regulation*
  • Genes, p53*
  • Mice
  • Mice, Transgenic
  • Models, Biological
  • Models, Genetic
  • Radiation Tolerance*
  • Radiation*
  • Spleen / metabolism
  • Tumor Suppressor Protein p53 / biosynthesis*
  • Tumor Suppressor Protein p53 / genetics*

Substances

  • Cyclin-Dependent Kinase Inhibitor p21
  • Tumor Suppressor Protein p53