Short-term endothelial progenitor cell colonies are composed of monocytes and do not acquire endothelial markers

Cytotherapy. 2007;9(1):14-22. doi: 10.1080/14653240601047726.

Abstract

Background: The aim of this study was to identify circulating endothelial progenitor cells (EPC) with colony-forming capacity and compare them with the monocytic-macrophage lineage.

Methods: Forty-two healthy donors were analyzed. EPC were cultured with VEGF and b-FGF. Sequential studies were performed on days +7 (colonies) +21 and +35. Monocytic cells were cultured using the same conditions as EPC until day +21 or alternatively by adding IGF.

Results: The number of EPC colonies was higher in BM than in mobilized or steady-state PB. Using EPC medium, monocytic cells formed cord-like structures but no colonies. However, colonies grew when IGF was added to the medium. By immunocytochemistry, colonies showed CD45, CD31 and lysozyme but no vWF. Colonies were CD4+, CD13+dim, CD14+, CD15++, CD16-/+dim, CD31+dim, CD33+dim, CD45+, CD105-/+dim, lysozyme+ and VE-cadherin+, and constantly negative for CD34, CD133 and KDR, when flow cytometry was used. The immunophenotype of pre-cultured and cultured monocytes was similar to that described for EPC.

Discussion: Our results suggest that the so-called 'EPC' obtained at 7 days of culture belong to the monocyte-macrophage lineage, as they share immunophenotypic and molecular features.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Antigens, CD / analysis
  • Biomarkers / analysis
  • Cells, Cultured
  • Endothelial Cells / cytology*
  • Endothelial Cells / drug effects
  • Endothelial Cells / metabolism
  • Female
  • Fibroblast Growth Factor 2 / pharmacology
  • Flow Cytometry
  • Gene Expression / drug effects
  • Humans
  • Immunohistochemistry
  • Male
  • Monocytes / cytology*
  • Monocytes / drug effects
  • Monocytes / metabolism
  • Reverse Transcriptase Polymerase Chain Reaction
  • Stem Cells / cytology*
  • Stem Cells / drug effects
  • Stem Cells / metabolism
  • Time Factors
  • Vascular Endothelial Growth Factor A / pharmacology
  • von Willebrand Factor / analysis
  • von Willebrand Factor / genetics

Substances

  • Antigens, CD
  • Biomarkers
  • Vascular Endothelial Growth Factor A
  • von Willebrand Factor
  • Fibroblast Growth Factor 2