Transcriptional repressor Gfi1 integrates cytokine-receptor signals controlling B-cell differentiation

PLoS One. 2007 Mar 21;2(3):e306. doi: 10.1371/journal.pone.0000306.

Abstract

Hematopoietic stem cell differentiation is specified by cytokines and transcription factors, but the mechanisms controlling instructive and permissive signalling networks are poorly understood. We provide evidence that CLP1-dependent IL7-receptor mediated B cell differentiation is critically controlled by the transcriptional repressor Gfi1. Gfi1-deficient progenitor B cells show global defects in IL7Ralpha-dependent signal cascades. Consequently, IL7-dependent trophic, proliferative and differentiation-inducing responses of progenitor B cells are perturbed. Gfi1 directly regulates expression levels of IL7Ralpha and indirectly controls STAT5 signalling via expression of SOCS3. Thus, Gfi1 selectively specifies IL7-dependent development of B cells from CLP1 progenitors, providing clues to the transcriptional networks integrating cytokine signals and lymphoid differentiation.

Publication types

  • Retracted Publication

MeSH terms

  • Animals
  • B-Lymphocytes / cytology*
  • B-Lymphocytes / drug effects
  • Bone Marrow Cells / cytology
  • Cell Differentiation / genetics
  • Cell Differentiation / physiology
  • Cell Division / drug effects
  • Interleukin-7 / pharmacology
  • Mice
  • Mice, Knockout
  • Proto-Oncogene Proteins / deficiency
  • Proto-Oncogene Proteins / genetics*
  • Receptors, Interleukin-7 / genetics*
  • Repressor Proteins / deficiency
  • Repressor Proteins / genetics*
  • Stem Cell Factor / pharmacology
  • Transcription, Genetic*

Substances

  • Gfi1b protein, mouse
  • Interleukin-7
  • Proto-Oncogene Proteins
  • Receptors, Interleukin-7
  • Repressor Proteins
  • Stem Cell Factor