[Kaposi's sarcoma-associated herpesvirus and mechanisms of oncogenesis]

Uirusu. 2006 Dec;56(2):209-18. doi: 10.2222/jsv.56.209.
[Article in Japanese]

Abstract

Kaposi's sarcoma-associated herpesvirus (KSHV, also known as human herpesvirus 8), is well known to be responsible for Kaposi's sarcoma, the most common AIDS-related cancer. KSHV is also associated with the B cell malignancies primary effusion lymphoma and multicentric Castleman's disease. Cellular signaling pathways regulate the proliferation and differentiation during normal development and a small number of signaling pathways are involved in tumors. KSHV utilize those pathways, such as pRb-E2F, Wnt and Notch pathways, to promote driving of cell cycle and to regulate their own life-cycles (i.e., latency and lytic cycle). This review focuses on signaling pathways which KSHV gene products manipulate and discusses their contributions to tomorigenesis and regulation of viral life-cycles.

Publication types

  • English Abstract
  • Review

MeSH terms

  • Animals
  • Cell Cycle
  • E2F Transcription Factors / physiology
  • Herpesvirus 8, Human* / genetics
  • Herpesvirus 8, Human* / growth & development
  • Herpesvirus 8, Human* / pathogenicity
  • Humans
  • Life Cycle Stages
  • Molecular Mimicry
  • Receptors, Notch / physiology
  • Sarcoma, Kaposi / virology*
  • Signal Transduction / genetics
  • Signal Transduction / physiology
  • Viral Proteins / physiology
  • Wnt Proteins / physiology

Substances

  • E2F Transcription Factors
  • Receptors, Notch
  • Viral Proteins
  • Wnt Proteins