Mutation of msh-2 in Neurospora crassa does not reduce the incidence of recombinants with multiple patches of donor chromosome sequence

Fungal Genet Biol. 2007 Jul;44(7):575-84. doi: 10.1016/j.fgb.2007.02.008. Epub 2007 Feb 28.

Abstract

The Neurospora homologue msh-2 of the Escherichia coli mismatch repair gene mutS was mutated by repeat-induced point mutation (RIP) of a 1.9-kb duplication covering 1661bp of the coding sequence and 302 bp 5' of the gene. msh-2(RIP-LK1) exhibited a mutator phenotype conferring a 17-fold increase in the frequency of spontaneous mitotic reversion of his-3 allele K458. In msh-2(RIP-LK1) homozygotes, recombination frequency at the his-3 locus increased up to 2.9-fold over that in msh-2(+) diploids. Progeny of crosses homozygous msh-2(RIP-LK1), like those from crosses homozygous msh-2(+) frequently had multiple patches of donor chromosome sequence, suggesting that patchiness in msh-2(+) crosses is not explained by incomplete repair of heteroduplex DNA by MSH-2. These findings are consistent with data from the analysis of events in a Neurospora translocation heterozygote that suggested multiple patches of donor chromosome sequence arising during recombination reflect multiple template switches during DNA repair synthesis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Base Pair Mismatch
  • Chromosomes, Fungal / genetics
  • Crossing Over, Genetic
  • DNA Mismatch Repair
  • DNA, Fungal / genetics*
  • Fungal Proteins / genetics*
  • Genes, Fungal
  • Molecular Sequence Data
  • MutS Homolog 2 Protein / genetics*
  • Neurospora crassa / genetics*
  • Point Mutation
  • Recombination, Genetic*
  • Sequence Alignment
  • Templates, Genetic

Substances

  • DNA, Fungal
  • Fungal Proteins
  • MutS Homolog 2 Protein