Calcitriol inhibits the growth of MHCC97 heptocellular cell lines by down-modulating c-met and ERK expressions

Liver Int. 2007 Jun;27(5):700-7. doi: 10.1111/j.1478-3231.2007.01487.x.

Abstract

Background: Previous studies have demonstrated that Calcitriol or its analogs has an anti-tumour activity. This study was designed to determine the effect and mechanism of Calcitriol on MHCC-97 heptocellular cell lines.

Methods: MHCC97 cell lines were treated with Calcitriol of 10(-6) approximately 10(-9) M concentration and with Calcitriol in lipiodol ultra-fluid (LUF) respectively. The conditions of cell proliferation were analyzed by MTT method. The cell apoptosis and cycle were analyzed by using a flow cytometer. Hepatocyte growth factor (HGF) concentration in the cell supernatant was measured by using ELISA method. C-met and vitamin D receptor (VDR) mRNA in cells were determined by using RT-PCR method. VDR and ERK(1/2) proteins were determined by using Western Blotting method.

Results: Calcitriol inhibited the proliferation of MHCC97 cell lines with an accumulation of cells in the G(0)/G(1) phase and reduction of cells in S phase. Calcitriol dissolved in LUF resulted in a better and longer inhibitive effect on the cell lines than Calcitriol alone. MHCC97 cell lines secreted HGF and expressed c-met mRNA and ERK(1/2) proteins abundantly. Calcitriol remarkably inhibited the expressions of c-met mRNA and ERK(1/2) proteins.

Conclusion: Calcitriol inhibits the growth of MHCC-97 heptocellular cell lines by down modulating c-met and ERK expressions.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / drug effects
  • Calcitriol / pharmacology*
  • Calcitriol / therapeutic use
  • Carcinoma, Hepatocellular / drug therapy*
  • Cell Cycle / drug effects
  • Cell Line
  • Cell Proliferation / drug effects*
  • Extracellular Signal-Regulated MAP Kinases / metabolism
  • Gene Expression / drug effects
  • Hepatocyte Growth Factor / metabolism
  • Humans
  • Liver Neoplasms / drug therapy*
  • Proto-Oncogene Proteins c-met / metabolism
  • Receptors, Calcitriol / metabolism
  • Vitamins / pharmacology*
  • Vitamins / therapeutic use

Substances

  • Receptors, Calcitriol
  • Vitamins
  • Hepatocyte Growth Factor
  • Proto-Oncogene Proteins c-met
  • Extracellular Signal-Regulated MAP Kinases
  • Calcitriol