NADH dehydrogenase subunit-2 237 Leu/Met polymorphism modifies the effects of alcohol consumption on risk for hypertension in middle-aged Japanese men

Hypertens Res. 2007 Mar;30(3):213-8. doi: 10.1291/hypres.30.213.

Abstract

NADH dehydrogenase subunit-2 237 leucine/methionine (ND2-237 Leu/Met) polymorphism is associated with longevity in the Japanese population, and the ND2-237Met genotype may exert antiatherogenic effects. To investigate whether ND2-237 Leu/Met polymorphism is associated with risk of hypertension, we conducted a cross-sectional study of 398 Japanese male subjects. The frequency of hypertension was significantly higher in ND2-237Leu genotypic men than in ND2-237Met genotypic men. On analysis of covariance, the interaction between ND2-237 Leu/Met polymorphism and habitual drinking was significantly associated with both systolic blood pressure and diastolic blood pressure. Multiple logistic regression analysis revealed that the ND2-237Met genotype, particularly in younger subjects (age <60 years), had a lower odds ratio for hypertension than the ND2-237Leu genotype. Moreover, the association of ND2-237 Leu/Met polymorphism with hypertension may depend on the frequency of alcohol consumption. The odds ratio for hypertension was significantly higher in daily drinkers with ND2-237Leu when compared with non- or ex-drinkers with ND2-237Leu. However, the association between the ND2-237Met genotype and hypertension may not depend on the frequency of alcohol consumption. The present results suggest that ND2-237 Leu/Met polymorphism is associated with hypertension and that modification of hypertension risk is dependent on alcohol consumption in middle-aged Japanese men.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alcohol Drinking / genetics*
  • Alcohol Drinking / physiopathology
  • Asian People / genetics
  • Blood Pressure / physiology
  • Cross-Sectional Studies
  • Genetic Predisposition to Disease
  • Genotype
  • Humans
  • Hypertension / epidemiology
  • Hypertension / genetics*
  • Hypertension / physiopathology
  • Japan / epidemiology
  • Leucine / genetics*
  • Male
  • Methionine / genetics*
  • Middle Aged
  • NADH Dehydrogenase / genetics*
  • NADH Dehydrogenase / metabolism
  • Polymorphism, Restriction Fragment Length*
  • Protein Subunits
  • Risk Factors

Substances

  • Protein Subunits
  • Methionine
  • NADH Dehydrogenase
  • NADH dehydrogenase subunit 2, human
  • Leucine