Abstract
Fosmidomycin, which acts through inhibition of 1-deoxy-D-xylulose phosphate reductoisomerase (DXR) in the non-mevalonate pathway, represents a valuable recent addition to the armamentarium against uncomplicated malaria. In this paper, we describe the synthesis and biological evaluation of E- and Z-alpha,beta-unsaturated alpha-aryl-substituted analogues of FR900098, a fosmidomycin congener, utilizing a Stille or a Suzuki coupling to introduce the aryl group. In contrast with our expectations based on the promising activity earlier observed for several alpha-substituted fosmidomycin analogues, all synthesized analogues exhibited much lower binding affinity for DXR than fosmidomycin.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Aldose-Ketose Isomerases / antagonists & inhibitors*
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Animals
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Antimalarials / chemical synthesis*
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Antimalarials / pharmacology
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Chemistry, Pharmaceutical / methods*
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Drug Design
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Drug Evaluation, Preclinical
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Enzyme Inhibitors / pharmacology*
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Fosfomycin / analogs & derivatives*
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Fosfomycin / chemical synthesis
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Fosfomycin / chemistry
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Inhibitory Concentration 50
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Malaria / drug therapy*
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Models, Chemical
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Molecular Structure
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Multienzyme Complexes / antagonists & inhibitors*
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Oxidoreductases / antagonists & inhibitors*
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Plasmodium falciparum / enzymology
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Structure-Activity Relationship
Substances
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Antimalarials
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Enzyme Inhibitors
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Multienzyme Complexes
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Fosfomycin
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fosmidomycin
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Oxidoreductases
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1-deoxy-D-xylulose 5-phosphate reductoisomerase
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Aldose-Ketose Isomerases