Iron sucrose causes greater proteinuria than ferric gluconate in non-dialysis chronic kidney disease

Kidney Int. 2007 Sep;72(5):638-42. doi: 10.1038/sj.ki.5002422. Epub 2007 Jul 11.

Abstract

Non-dextran intravenous (i.v.) iron preparations seem to differentially affect proteinuria in patients with chronic kidney disease. To study effects of ferric gluconate and iron sucrose on proteinuria, we conducted a crossover trial in 12 patients with stage 3-4 chronic kidney disease. These patients were randomized to receive the same dose of either drug 1 week apart. Urine samples were obtained immediately before and at frequent intervals after the drug. The urine total protein/creatinine ratio was significantly greater after iron sucrose than ferric gluconate treatment with the effect noted within 15 min post-infusion. Furthermore, when iron sucrose was given first, a significantly greater protein/creatinine ratio was seen subsequently with ferric gluconate than with the reverse order of treatment. The urine albumin/creatinine ratio was also significantly greater with iron sucrose than with ferric gluconate. There was no significant difference, however, between the two i.v. irons in the measured urine N-acetyl-beta-D-glucosaminidase/creatinine ratio. Although our study showed that acutely, iron sucrose increased proteinuria, the long-term effects of repeated i.v. non-dextran iron on kidney function requires further study.

Publication types

  • Randomized Controlled Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Cross-Over Studies
  • Female
  • Ferric Compounds / administration & dosage*
  • Ferric Compounds / therapeutic use
  • Ferric Oxide, Saccharated
  • Glucaric Acid
  • Humans
  • Male
  • Middle Aged
  • Proteinuria / drug therapy*
  • Renal Insufficiency, Chronic / drug therapy*
  • Treatment Outcome

Substances

  • Ferric Compounds
  • Ferric Oxide, Saccharated
  • Glucaric Acid
  • ferric gluconate