A presenilin 1 mutation (L420R) in a family with early onset Alzheimer disease, seizures and cotton wool plaques, but not spastic paraparesis

Neuropathology. 2007 Jun;27(3):228-32. doi: 10.1111/j.1440-1789.2007.00766.x.

Abstract

Over 100 mutations in the presenilin-1 gene (PSEN1) have been shown to result in familial early onset Alzheimer disease (EOAD), but only a relatively few give rise to plaques with an appearance like cotton wool (CWP) and/or spastic paraparesis (SP). A family with EOAD, seizures and CWP was investigated by neuropathological study and DNA sequencing of the PSEN1 gene. Abeta was identified in leptomeningeal vessels and in cerebral plaques. A single point mutation, p.L420R (g.1508T > G) that gives rise to a missense mutation in the eighth transmembrane (TM8) domain of PS1 was identified in two affected members of the family. p.L420R (g.1508T > G) is the mutation responsible for EOAD, seizures and CWP without SP in this family.

Publication types

  • Case Reports

MeSH terms

  • Alzheimer Disease / genetics*
  • Alzheimer Disease / pathology
  • Alzheimer Disease / physiopathology
  • Amyloid beta-Peptides / metabolism
  • Arthritis, Rheumatoid / pathology
  • Brain / pathology
  • DNA Mutational Analysis
  • Diabetes Mellitus, Type 2 / pathology
  • Female
  • Humans
  • Hypertension / pathology
  • Immunohistochemistry
  • Male
  • Mutation, Missense
  • Paraparesis, Spastic / pathology*
  • Pedigree
  • Plaque, Amyloid / pathology*
  • Point Mutation
  • Presenilin-1 / chemistry
  • Presenilin-1 / genetics*
  • Seizures / etiology*

Substances

  • Amyloid beta-Peptides
  • Presenilin-1