Marked genetic differences in the regulation of blood glucose under immune and restraint stress in mice reveals a wide range of corticosensitivity

J Neuroimmunol. 2007 Sep;189(1-2):59-68. doi: 10.1016/j.jneuroim.2007.06.019. Epub 2007 Jul 20.

Abstract

Male and female mice from different control strains (C57BL/6, DBA/2, BALB/c) and the nonobese diabetic (NOD) strain, a spontaneous model of type 1 diabetes, were subjected to various stressors (restraint, lipopolysaccharide or interleukin-1 injection). Significant differences were measured among strains in blood glucose, insulin and corticosterone levels and, for restraint, IL-6. Addition of dexamethasone, a glucocorticoid receptor agonist, to inhibit the expression of several proteins by LPS-stimulated bone marrow-derived macrophages in vitro showed a gradient among control strains: C57BL/6>DBA/2>BALB/c corroborating the pattern of corticosensitivity suggested by their stress-induced glucose responses at the systemic level.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Analysis of Variance
  • Animals
  • Animals, Inbred Strains
  • Blood Glucose / metabolism*
  • Cells, Cultured
  • Corticosterone / metabolism*
  • Cytokines / metabolism
  • Dexamethasone / pharmacology
  • Diabetes Mellitus, Type 1 / blood
  • Diabetes Mellitus, Type 1 / physiopathology
  • Enzyme-Linked Immunosorbent Assay / methods
  • Female
  • Insulin / blood
  • Interleukin-1 / adverse effects
  • Interleukin-6 / metabolism
  • Macrophages / drug effects
  • Macrophages / physiology
  • Male
  • Mice
  • Nitric Oxide Synthase Type II / metabolism
  • Polysaccharides / adverse effects
  • Restraint, Physical / methods
  • Species Specificity
  • Stress, Physiological / etiology
  • Stress, Physiological / immunology*
  • Stress, Physiological / physiopathology*

Substances

  • Blood Glucose
  • Cytokines
  • Insulin
  • Interleukin-1
  • Interleukin-6
  • Polysaccharides
  • Dexamethasone
  • Nitric Oxide Synthase Type II
  • Corticosterone