Endothelial NF-kappaB as a mediator of kidney damage: the missing link between systemic vascular and renal disease?

Circ Res. 2007 Aug 3;101(3):227-9. doi: 10.1161/CIRCRESAHA.107.158295.
No abstract available

Publication types

  • Editorial
  • Research Support, N.I.H., Extramural
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Comment

MeSH terms

  • Albuminuria / etiology
  • Albuminuria / prevention & control
  • Angiotensin II / toxicity
  • Animals
  • Atherosclerosis / physiopathology
  • Endothelium, Vascular / metabolism*
  • Fibrosis
  • Gemfibrozil / pharmacology
  • Gemfibrozil / therapeutic use
  • Humans
  • Hypertension / complications*
  • Hypertension / metabolism
  • I-kappa B Proteins / genetics
  • I-kappa B Proteins / physiology
  • Inflammation / physiopathology
  • Kidney Diseases / etiology*
  • Kidney Diseases / prevention & control
  • Mice
  • Models, Cardiovascular
  • NF-KappaB Inhibitor alpha
  • NF-kappa B / antagonists & inhibitors
  • NF-kappa B / physiology*
  • Nitric Oxide / physiology
  • Nitric Oxide Synthase Type III / antagonists & inhibitors
  • Organ Specificity
  • Sodium Chloride, Dietary / toxicity

Substances

  • I-kappa B Proteins
  • NF-kappa B
  • NFKBIA protein, human
  • Nfkbia protein, mouse
  • Sodium Chloride, Dietary
  • Angiotensin II
  • NF-KappaB Inhibitor alpha
  • Nitric Oxide
  • Nitric Oxide Synthase Type III
  • Gemfibrozil