A comparative analysis of lichen sclerosus of the vulva and lichen sclerosus that evolves to vulvar squamous cell carcinoma

Am J Obstet Gynecol. 2007 Dec;197(6):592.e1-5. doi: 10.1016/j.ajog.2007.04.003. Epub 2007 Aug 21.

Abstract

Objective: The aim of this study was to determine whether the premalignant change in lichen sclerosus (LS) could be identified with immunohistochemical analyses.

Study design: Eight cases of histologically diagnosed vulvar LS, which showed, after a period of 10 months-9 years, an evolution to carcinoma of the vulva that was histologically documented, were compared with 8 cases of vulvar LS, for which follow-up information was available for at least 9 years. The proliferative index and the expression of tumor suppressors p16 and p53 were analyzed.

Results: The difference of MIB1 labeling index of evolving or unchanged LS cases was significant (P = .005). The difference in the p53 of evolving or unchanged LS cases shows a trend towards association (P = .08). Both LS cases (evolving or unchanged) did not show p16 positive staining.

Conclusion: The evaluation of MIB1 and p53 may identify those vulvar LS cases with a high likelihood of evolving into squamous cell carcinoma, which would need careful periodic checks or adjunctive biopsies. The study must be confirmed by a larger number of cases to substantiate this observation.

Publication types

  • Comparative Study

MeSH terms

  • Adult
  • Aged
  • Carcinoma, Squamous Cell / diagnosis*
  • Carcinoma, Squamous Cell / etiology
  • Cell Proliferation
  • Female
  • Genes, p53 / physiology
  • Humans
  • Immunohistochemistry
  • Middle Aged
  • Precancerous Conditions / complications
  • Precancerous Conditions / diagnosis*
  • Ubiquitin-Protein Ligases / analysis
  • Ubiquitin-Protein Ligases / biosynthesis
  • Vulvar Lichen Sclerosus / complications
  • Vulvar Lichen Sclerosus / diagnosis*
  • Vulvar Neoplasms / diagnosis*
  • Vulvar Neoplasms / etiology

Substances

  • MIB1 ligase, human
  • Ubiquitin-Protein Ligases