Platelet aggregation by strains of enterococci

Microbiol Immunol. 1991;35(11):933-42. doi: 10.1111/j.1348-0421.1991.tb01615.x.

Abstract

The platelet aggregation capability of whole cells of Enterococcus faecalis, E. faecium and E. avium was tested. The optimum ratios of bacteria to platelets in E. faecalis (strain SMU-37), E. faecium (strain SMU-138) and E. avium (strain SMU-197) were 1.0, 1.2 and 2.0, respectively. During the platelet aggregation induced by the three strains of enterococci, 65-69% of total serotonin was released. The aggregation was totally inhibited by ethylenediaminetetraacetate (10 mM) and apyrase (1 mg/ml), while no effect was shown by aspirin (10 mM), indomethacin (10 mM) and quinacrine (1 mM). By pretreatment of platelet-poor plasma with heat (56 C, 30 min) or zymosan, the reactivities with platelets of each strain of species were markedly diminished. These results suggest that enterococci-induced platelet aggregation was an ion-dependent, cyclooxygenase-insensitive event, and plasma component(s) was (were) required for the reaction.

MeSH terms

  • Animals
  • Arachidonic Acid / metabolism
  • Enterococcus / physiology*
  • In Vitro Techniques
  • Plasma / physiology
  • Platelet Aggregation / physiology*
  • Rabbits

Substances

  • Arachidonic Acid