Corepressor CtBP and nuclear speckle protein Pnn/DRS differentially modulate transcription and splicing of the E-cadherin gene

Mol Cell Biol. 2008 Mar;28(5):1584-95. doi: 10.1128/MCB.00421-07. Epub 2007 Dec 17.

Abstract

CtBP is a transcriptional corepressor with tumorigenic potential that targets the promoter of the tumor suppressor gene E-cadherin. Pnn/DRS (Pnn) is a "nuclear speckle"-associated protein involved in mRNA processing as well as transcriptional regulation of E-cadherin via its binding to CtBP. Here, we show that CtBP can recruit Pnn to CtBP-associated complexes, resulting in Pnn-dependent chromatin remodeling at the E-cadherin promoter. In addition, CtBP and Pnn can differentially modulate E-cadherin mRNA splicing, with polymerase II serving as an interface in this event. Therefore, the Pnn/CtBP functional interplay represents a novel mechanism linking the corepressor CtBP and Pnn to the transcription-coupled mRNA splicing of a major tumor suppressor gene. Our findings implicate the existence of the molecular switches involved in tumorigenesis, which coordinate promoter-specific events and mRNA processing, by serving as bridging elements between the regulatory complexes both at gene promoters and within the mRNA splicing machineries.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Alcohol Oxidoreductases / genetics
  • Alcohol Oxidoreductases / metabolism*
  • Cadherins / genetics*
  • Cell Adhesion Molecules / genetics
  • Cell Adhesion Molecules / metabolism*
  • Cell Line
  • Chromatin Assembly and Disassembly
  • Chromatin Immunoprecipitation
  • DNA, Complementary
  • DNA-Binding Proteins / genetics
  • DNA-Binding Proteins / metabolism*
  • Gene Expression Regulation
  • Genes, Reporter
  • Genetic Vectors
  • Green Fluorescent Proteins / metabolism
  • HeLa Cells
  • Hemagglutinins / metabolism
  • Humans
  • Kidney / cytology
  • Luciferases / metabolism
  • Models, Biological
  • Nuclear Proteins / genetics
  • Nuclear Proteins / metabolism*
  • Precipitin Tests
  • Promoter Regions, Genetic
  • Protein Binding
  • RNA Interference
  • RNA Polymerase II / metabolism
  • RNA Splicing*
  • RNA, Messenger / metabolism
  • Recombinant Fusion Proteins / metabolism
  • Repressor Proteins / metabolism
  • Transcription, Genetic*
  • Transfection

Substances

  • Cadherins
  • Cell Adhesion Molecules
  • DNA, Complementary
  • DNA-Binding Proteins
  • Hemagglutinins
  • Nuclear Proteins
  • PNN protein, human
  • RNA, Messenger
  • Recombinant Fusion Proteins
  • Repressor Proteins
  • Green Fluorescent Proteins
  • Alcohol Oxidoreductases
  • C-terminal binding protein
  • Luciferases
  • RNA Polymerase II