[Effect of lycopene on proliferation and cell cycle of hormone refractory prostate cancer PC-3 cell line]

Wei Sheng Yan Jiu. 2007 Sep;36(5):575-8.
[Article in Chinese]

Abstract

Objective: To investigate the effects of lycopene on proliferation of human prostate cancer cell line PC-3 and explore its effecting mechanism.

Methods: The proliferation of PC-3 cells were analyzed by MTT and H3-TdR incorporation. The effects of cell cycle and apoptosis of synchronized cells were observed through flow cytometry. RT-PCR methods were used to explore the mRNA expression level of cyclin D1, bcl-2, bax.

Results: The growth and DNA synthesis of PC-3 cell were inhibited with the lycopene concentration increased, and lycopene also could change the cell cycle distribution, i. e. increasing the proportion of G0/G1 phase and descending the proportion of S and G2/M phase, and induce the apoptosis. RT-PCR analysis showed that the mRNA expression level of cyclin D1 and bcl-2 were down regulated, while the level of bax was up regulated.

Conclusion: Lycopene can induce apoptosis of PC-3, change the cell cycle distribution and downregulate the expression of cyclin D1 and bcl-2 and upregulate the expression of bax and then restrain cell proliferation.

MeSH terms

  • Antineoplastic Agents, Phytogenic / pharmacology*
  • Carotenoids / pharmacology*
  • Cell Cycle / drug effects*
  • Cell Line, Tumor
  • Cell Proliferation / drug effects*
  • Cyclin D1 / genetics
  • Cyclin D1 / metabolism
  • Humans
  • Lycopene
  • Male
  • Prostatic Neoplasms / pathology*
  • Proto-Oncogene Proteins c-bcl-2 / genetics
  • Proto-Oncogene Proteins c-bcl-2 / metabolism
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • bcl-2-Associated X Protein / genetics
  • bcl-2-Associated X Protein / metabolism

Substances

  • Antineoplastic Agents, Phytogenic
  • Proto-Oncogene Proteins c-bcl-2
  • RNA, Messenger
  • bcl-2-Associated X Protein
  • Cyclin D1
  • Carotenoids
  • Lycopene