CYP3A5 genotype is associated with longer patient survival after kidney transplantation and long-term treatment with cyclosporine

Pharmacogenomics J. 2008 Dec;8(6):416-22. doi: 10.1038/sj.tpj.6500488. Epub 2008 Jan 8.

Abstract

The CYP3A5*1 allele has been linked to high expression of CYP3A5 and metabolism of cyclosporine. We evaluated the role of CYP3A5*1 for long-term survival in renal transplant patients in a cohort of 399 patients who underwent cadaveric or living donor kidney allograft transplantation. All patients were treated with a similar cyclosporine-based immunosuppressive maintenance therapy protocol. The mean duration of follow-up was 8.6+/-3.7 years. In univariate survival analysis, the presence of the CYP3A5*1 allele in recipients significantly increased patient survival P=0.028 (log-rank), resulting in a hazard ratio (HR) of 0.52 (95% CI=0.29-0.94). When the presence of the CYP3A5*1 allele was included in multivariate Cox regression analyses accounting for major risk factors for patient death, CYP3A5*1 still conferred a protective effect. Further, haplotype analysis at the CYP3A5 locus confirmed that CYP3A5*1 might indeed be responsible for this survival benefit.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cohort Studies
  • Cyclosporine / therapeutic use*
  • Cytochrome P-450 CYP3A / genetics*
  • Genotype
  • Humans
  • Immunosuppressive Agents / therapeutic use*
  • Kidney Transplantation*
  • Survival Analysis

Substances

  • Immunosuppressive Agents
  • Cyclosporine
  • CYP3A5 protein, human
  • Cytochrome P-450 CYP3A