Increased vascular cell adhesion molecule-1 was associated with impaired endothelium-dependent relaxation of cerebral and carotid arteries in simulated microgravity rats

J Physiol Sci. 2008 Feb;58(1):67-73. doi: 10.2170/physiolsci.RP010707. Epub 2008 Jan 29.

Abstract

The aim of the present study was to investigate whether an expression of vascular cell adhesion molecule-1 (VCAM-1) was upregulated in 3-week simulated microgravity rat cerebral and carotid arteries and whether impaired endothelium-dependent relaxation was concomitant with VCAM-1 expression. Male Sprague-Dawley rats were randomly divided into control (CON) and hindlimb unweighting (HU) groups. After 3 weeks, the expression of the VCAM-1 protein and the vasodilatation of the basilar artery and common carotid artery were determined. Immunohistochemical results revealed positive staining of VCAM-1 on endothelial cells in these arteries from HU compared with CON rats. Western blot analysis confirmed an upregulated expression of VCAM-1 protein in these arteries from HU rats. Acetylcholine induced concentration-dependent vasodilatation in all artery rings, but with significantly smaller amplitude in the basilar artery (P < 0.01) and the common carotid artery (P < 0.05) from HU than those from CON rats. The data suggested that the expression of VCAM-1 protein was upregulated in cerebral and common carotid arteries of simulated microgravity rats, and the upregulation of VCAM-1 may contribute to impaired endothelium-dependent relaxation in simulated microgravity rat vasculature.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetylcholine / pharmacology
  • Animals
  • Basilar Artery / physiology*
  • Carotid Artery, Common / physiology*
  • Endothelium, Vascular / metabolism*
  • Hindlimb
  • Immunohistochemistry
  • Isometric Contraction / drug effects
  • Isometric Contraction / physiology
  • Male
  • Nitroprusside / pharmacology
  • Rats
  • Rats, Sprague-Dawley
  • Up-Regulation / physiology
  • Vascular Cell Adhesion Molecule-1 / metabolism*
  • Vasculitis / metabolism*
  • Vasculitis / physiopathology
  • Vasodilation / drug effects
  • Vasodilation / physiology*
  • Vasodilator Agents / pharmacology
  • Weightlessness Simulation*

Substances

  • Vascular Cell Adhesion Molecule-1
  • Vasodilator Agents
  • Nitroprusside
  • Acetylcholine