Functional properties of cells obtained from human cord blood CD34+ stem cells and mouse cardiac myocytes in coculture

Am J Physiol Heart Circ Physiol. 2008 Apr;294(4):H1541-9. doi: 10.1152/ajpheart.01285.2007. Epub 2008 Jan 25.

Abstract

Prior in vitro studies suggested that different types of hematopoietic stem cells may differentiate into cardiomyocytes. The present work examined whether human CD34(+) cells from the human umbilical cord blood (hUCB), cocultured with neonatal mouse cardiomyocytes, acquire the functional properties of myocardial cells and express human cardiac genes. hUCB CD34(+) cells were cocultured onto cardiomyocytes following an infection with a lentivirus-encoding enhanced green fluorescent protein (EGFP). After 7 days, mononucleated EGFP(+) cells were tested for their electrophysiological features by patch clamp and for cytosolic [Ca(2+)] ([Ca(2+)](i)) homeostasis by [Ca(2+)](i) imaging of X-rhod1-loaded cells. Human Nkx2.5 and GATA-4 expression was examined in cocultured cell populations by real-time RT-PCR. EGFP(+) cells were connected to surrounding cells by gap junctions, acquired electrophysiological properties similar to those of cardiomyocytes, and showed action potential-associated [Ca(2+)](i) transients. These cells also exhibited spontaneous sarcoplasmic reticulum [Ca(2+)](i) oscillations and the associated membrane potential depolarization. However, RT-PCR of both cell populations showed no upregulation of human-specific cardiac genes. In conclusion, under our experimental conditions, hUCB CD34(+) cells cocultured with murine cardiomyocytes formed cells that exhibited excitation-contraction coupling features similar to those of cardiomyocytes. However, the expression of human-specific cardiac genes was undetectable by RT-PCR.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Animals, Newborn
  • Antigens, CD34 / analysis*
  • Calcium / metabolism
  • Cell Communication* / genetics
  • Cell Differentiation* / genetics
  • Cell Shape
  • Cell Transdifferentiation
  • Cells, Cultured
  • Coculture Techniques
  • Fetal Blood / cytology
  • Fetal Blood / immunology
  • Fetal Blood / metabolism*
  • GATA4 Transcription Factor / genetics
  • GATA4 Transcription Factor / metabolism
  • Gap Junctions / metabolism
  • Genes, Reporter
  • Genetic Vectors
  • Green Fluorescent Proteins / biosynthesis
  • Green Fluorescent Proteins / genetics
  • Homeobox Protein Nkx-2.5
  • Homeodomain Proteins / genetics
  • Homeodomain Proteins / metabolism
  • Humans
  • Lentivirus / genetics
  • Membrane Potentials
  • Mice
  • Myocytes, Cardiac / metabolism*
  • Patch-Clamp Techniques
  • RNA, Messenger / metabolism
  • Reverse Transcriptase Polymerase Chain Reaction
  • Sarcoplasmic Reticulum / metabolism
  • Stem Cells / immunology
  • Stem Cells / metabolism
  • Stem Cells / physiology*
  • Time Factors
  • Transcription Factors / genetics
  • Transcription Factors / metabolism
  • Transduction, Genetic

Substances

  • Antigens, CD34
  • GATA4 Transcription Factor
  • GATA4 protein, human
  • Homeobox Protein Nkx-2.5
  • Homeodomain Proteins
  • NKX2-5 protein, human
  • RNA, Messenger
  • Transcription Factors
  • enhanced green fluorescent protein
  • Green Fluorescent Proteins
  • Calcium