The association of myeloperoxidase promoter polymorphism with carotid atherosclerosis is abolished in patients with type 2 diabetes

Clin Biochem. 2008 May;41(7-8):532-7. doi: 10.1016/j.clinbiochem.2008.01.017. Epub 2008 Feb 6.

Abstract

Objectives: Type 2 diabetes mellitus (DM) enhances the development of atherosclerosis and reduces the activity of the oxidative myeloperoxidase (MPO) enzyme. MPO gene has a functional promoter polymorphism -463G/A which leads to high- (GG) and low-expression (AG, AA) genotypes.

Design and methods: We studied the association of MPO polymorphism with carotid artery intima-media thickness (IMT) in 198 randomly selected Finnish men of Caucasian origin, 161 non-diabetics and 37 with type 2 DM. Their carotid IMT was measured by high-resolution ultrasonography, and the overall mean IMT value was calculated. MPO genotypes were determined by the PCR-RFLP method.

Results: We found significant MPO genotype-by-study-group (control/DM) interactions with the overall mean IMT and internal carotid IMT (p=0.05 and p=0.04, respectively). Among non-diabetic subjects, the overall carotid IMT was 7.3% higher in subjects with the low-activity genotype when compared to the high-activity (G/G) group. The results remained significant after adjustment for total cholesterol and smoking (p=0.015). No similar genotypic association was found for the subjects with type 2 DM.

Conclusions: This data suggests that in subjects with normal glucose metabolism, MPO gene variation may modify the carotid artery IMT.

Publication types

  • Comparative Study
  • Multicenter Study
  • Randomized Controlled Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Blood Glucose / metabolism
  • Carotid Artery Diseases / complications
  • Carotid Artery Diseases / enzymology*
  • Carotid Artery Diseases / genetics*
  • Cohort Studies
  • Diabetes Mellitus, Type 2 / complications
  • Diabetes Mellitus, Type 2 / enzymology*
  • Diabetes Mellitus, Type 2 / genetics*
  • Genetic Variation
  • Genotype
  • Humans
  • Male
  • Middle Aged
  • Peroxidase / antagonists & inhibitors*
  • Peroxidase / genetics*
  • Peroxidase / physiology
  • Polymorphism, Genetic / genetics*
  • Promoter Regions, Genetic / genetics*

Substances

  • Blood Glucose
  • Peroxidase