Biologic study of the effects of octreotide-LAR on growth hormone in unresectable and metastatic hepatocellular carcinoma

Clin Adv Hematol Oncol. 2008 Jan;6(1):44-54.

Abstract

Background: Animal models suggest that growth hormone participates in hepatocarcinogenesis.

Objective: To correlate the effect of octreotide long-acting release (LAR) on insulin-like growth factor-I (IGF-I) and -II (IGF-II) with response and survival in patients with unresectable and metastatic hepatocellular carcinoma.

Methods: We conducted a phase II, single-institution trial of octreotide-LAR (30 mg intramuscularly every 4 weeks) in 15 patients while monitoring serum IGF-I and -II levels.

Results: Patients (median CLIP score 2, Okuda stage II, and ECOG performance status 1) were treated for a median of 2.0 cycles. No responses occurred. Median overall survival was 116 days (range, 27-937 days) and median progression-free survival was 60 days (range, 27-444 days). One patient had prolonged stable disease (16 months). There were no grade 4 and four grade 3 toxicities: abdominal cramping, elevated creatinine, diarrhea, and dyspnea. Median serum IGF-I decreased from baseline (42.2 ng/mL; range, 14.2-109 ng/mL) to day 29 (27.9 ng/mL; range, 5.7-71.1 ng/mL), and median serum IGF-II decreased from baseline (25,000 ng/mL; range, 12,400-93,600 ng/mL) to day 29 (18,400 ng/mL; range, 4,061-79,400 ng/mL; 2-sided P<.006 and P<.04, respectively; Wilcoxon signed rank test). This suppression did not correlate with clinical activity. Baseline serum IGF-I >30 ng/mL was associated with greater progression-free survival and overall survival (P=.0005 and P=.0173, respectively; 2-sided log-rank test).

Conclusions: Octreotide-LAR lowered serum IGF-I and -II levels; however, this lowering did not correlate with clinical activity. There were no responses, and progression-free survival and overall survival were similar to historical patients not on treatment. Baseline serum IGF-I predicted prognosis.

Publication types

  • Clinical Trial, Phase II
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Antineoplastic Agents, Hormonal / pharmacology*
  • Carcinoma, Hepatocellular / drug therapy*
  • Delayed-Action Preparations
  • Female
  • Humans
  • Insulin-Like Growth Factor I / drug effects*
  • Insulin-Like Growth Factor II / drug effects*
  • Kaplan-Meier Estimate
  • Liver Neoplasms / drug therapy*
  • Male
  • Middle Aged
  • Neoplasm Metastasis / drug therapy
  • Octreotide / pharmacology*
  • Prognosis

Substances

  • Antineoplastic Agents, Hormonal
  • Delayed-Action Preparations
  • Insulin-Like Growth Factor I
  • Insulin-Like Growth Factor II
  • Octreotide