The novel gene AngRem104 downregulates glucocorticoid receptor expression and activates NF-kappaB in human mesangial cells

Biochem Biophys Res Commun. 2008 May 16;369(4):1057-60. doi: 10.1016/j.bbrc.2008.02.148. Epub 2008 Mar 10.

Abstract

AngRem104 [angiotensin II (Ang II)-related genes in human mesangial cells (MCs), clone104], a novel gene in human MCs induced by Ang II, was previously identified in human MCs and found to interact with several proteins. The current study used a yeast two-hybrid system and co-immunoprecipitation to investigate the interaction between AngRem104 and glucocorticoid receptor (GR) AF-1-specific elongation factor (GR-EF). GR expression was downregulated and the number of MCs positive for activated nuclear factor kappaB (NF-kappaB) was increased when AngRem104 was overexpressed. Transfection with antisense AngRem104 vector resulted in the upregulation of GR protein and reduced numbers of MCs with activated NF-kappaB. These results indicate that the novel gene AngRem104 is involved in the in vivo regulation of GR expression and the activation of NF-kappaB through interaction with GR-EF in human MCs.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cells, Cultured
  • Down-Regulation
  • Glomerular Mesangium / cytology
  • Glomerular Mesangium / metabolism*
  • Humans
  • NF-kappa B / metabolism*
  • Nuclear Proteins / antagonists & inhibitors
  • Nuclear Proteins / genetics
  • Nuclear Proteins / metabolism*
  • Peptide Elongation Factor 1 / metabolism*
  • Receptors, Glucocorticoid / metabolism*
  • Two-Hybrid System Techniques

Substances

  • EEF1A1 protein, human
  • NF-kappa B
  • Nuclear Proteins
  • Peptide Elongation Factor 1
  • Receptors, Glucocorticoid