Gene expression-based screening identifies microtubule inhibitors as inducers of PGC-1alpha and oxidative phosphorylation

Proc Natl Acad Sci U S A. 2008 Mar 25;105(12):4721-6. doi: 10.1073/pnas.0800979105. Epub 2008 Mar 17.

Abstract

The transcriptional coactivator PGC-1alpha is a potent regulator of several metabolic pathways, including, in particular, the activation of oxidative phosphorylation and mitochondrial biogenesis. Recent evidence suggests that increasing PGC-1alpha activity may have beneficial effects in various conditions, including muscular dystrophy, diabetes, and neurodegenerative diseases. We describe here a high-throughput screen to identify small molecules that induce PGC-1alpha expression in skeletal muscle cells. A number of drug classes are identified, including glucocorticoids, microtubule inhibitors, and protein synthesis inhibitors. These drugs induce PGC-1alpha mRNA, and the expression of a number of genes known to be regulated by PGC-1alpha. No induction of these target genes is seen in PGC-1alpha -/- cells, demonstrating that the drugs act through PGC-1alpha. These data demonstrate the feasibility of high-throughput screening for inducers of PGC-1alpha. Moreover, the data identify microtubule inhibitors and protein synthesis inhibitors as modulators of PGC-1alpha and oxidative phosphorylation.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Cells, Cultured
  • Drug Evaluation, Preclinical
  • Drug Synergism
  • Gene Expression Profiling*
  • Gene Expression Regulation / drug effects
  • Glucocorticoids / pharmacology
  • Mice
  • Mice, Inbred C57BL
  • Microtubules / drug effects*
  • Muscle Cells / drug effects
  • Muscle Cells / metabolism
  • Muscle, Skeletal / cytology
  • Muscle, Skeletal / drug effects
  • Muscle, Skeletal / metabolism
  • Oxidative Phosphorylation / drug effects*
  • Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha
  • Protein Biosynthesis / drug effects
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • Trans-Activators / genetics*
  • Trans-Activators / metabolism
  • Transcription Factors
  • Tubulin Modulators / analysis*
  • Tubulin Modulators / pharmacology*

Substances

  • Glucocorticoids
  • Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha
  • Ppargc1a protein, mouse
  • RNA, Messenger
  • Trans-Activators
  • Transcription Factors
  • Tubulin Modulators