Circumvention of glucocorticoid resistance in childhood leukemia

Leuk Res. 2008 Sep;32(9):1417-23. doi: 10.1016/j.leukres.2008.02.009. Epub 2008 Apr 18.

Abstract

In this study, we determined if in vitro resistance to prednisolone and dexamethasone could be circumvented by cortivazol or methylprednisolone, or reversed by meta-iodobenzylguanidine in pediatric lymphoblastic and myeloid leukemia. As there were strong correlations between the LC50 values (drug concentration inducing 50% leukemic cell kill, LCK) of the different glucocorticoids and median prednisolone/methylprednisolone, prednisolone/dexamethasone and prednisolone/cortivazol LC50 ratios did not differ between the leukemia subtypes, we conclude that none of the glucocorticoids had preferential anti-leukemic activity. Meta-iodobenzylguanidine however, partially reversed glucocorticoid resistance in 19% of the lymphoblastic leukemia samples.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Proliferation
  • Child
  • Dexamethasone / therapeutic use
  • Drug Resistance, Neoplasm*
  • Drug Screening Assays, Antitumor
  • Glucocorticoids / therapeutic use*
  • Humans
  • Leukemia, Myeloid, Acute / drug therapy*
  • Leukemia, Myeloid, Acute / pathology
  • Methylprednisolone / therapeutic use*
  • Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy*
  • Precursor Cell Lymphoblastic Leukemia-Lymphoma / pathology
  • Prednisolone / therapeutic use
  • Pregnatrienes / therapeutic use*
  • Tumor Cells, Cultured

Substances

  • Glucocorticoids
  • Pregnatrienes
  • Dexamethasone
  • Prednisolone
  • Methylprednisolone
  • cortivazol