Acute promyelocytic leukemia after mitoxantrone therapy for multiple sclerosis

Cancer Genet Cytogenet. 2008 Apr 15;182(2):126-9. doi: 10.1016/j.cancergencyto.2008.01.004.

Abstract

Mitoxantrone is a DNA-topoisomerase 2 inhibitor used as a single agent for treatment of relapsing-remitting or progressive multiple sclerosis (MS). We present here two patients treated with mitoxantrone for MS who subsequently developed acute promyelocytic leukemia (APL). These constitute, to our knowledge, the eighth and ninth reports of APL in patients treated with mitoxantrone for MS. Topoisomerase 2 inhibitors are associated with therapy-related acute myeloid leukemia (t-AML) with 11q23 abnormalities, but therapy-related APL (t-APL) is less common, and documentation of nine cases of t-APL after mitoxantrone therapy for MS suggests a specific association.

Publication types

  • Case Reports

MeSH terms

  • Antineoplastic Agents / adverse effects*
  • Chromosomes, Human, Pair 11 / genetics
  • Female
  • Humans
  • In Situ Hybridization, Fluorescence
  • Karyotyping
  • Leukemia, Promyelocytic, Acute / chemically induced*
  • Leukemia, Promyelocytic, Acute / drug therapy
  • Male
  • Middle Aged
  • Mitoxantrone / adverse effects*
  • Multiple Sclerosis / drug therapy*
  • Remission Induction
  • Translocation, Genetic

Substances

  • Antineoplastic Agents
  • Mitoxantrone