Autoproteolytic cleavage of recombinant 3C proteinase of hepatitis A virus

Virology. 1991 Jun;182(2):861-4. doi: 10.1016/0042-6822(91)90630-t.

Abstract

A hepatitis A virus cDNA fragment coding for the viral proteinase 3C was expressed as a chimeric protein fused in-frame to the C-terminus of beta-galactosidase. Following induction of the lac Z promoter, polypeptides of 150, 28, 26, and 16 kDa, all of which carry 3C antigenicity, were produced. The 28- and 26-kDa proteins were identified as autoproteolytic products of the fusion protein by determination of their N-terminal amino acid sequence. The 16-kDa protein arises from internal initiation. Following substitution of the 37 amino acids at the C-terminus of 3C, the autolytic activity was no longer observed. The recombinant proteinase did not show trans-activity when recombinant proteins of the P1 or P2 region were used as substrates. Antisera directed against recombinant 3C could not detect 3C or its precursors in HAV-infected cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 3C Viral Proteases
  • Amino Acid Sequence
  • Cloning, Molecular
  • Cysteine Endopeptidases / genetics
  • Cysteine Endopeptidases / immunology
  • Cysteine Endopeptidases / metabolism*
  • Hepatovirus / enzymology*
  • Hepatovirus / metabolism
  • Molecular Sequence Data
  • Peptides / immunology
  • Protein Processing, Post-Translational
  • Proteins / metabolism
  • Recombinant Proteins / immunology
  • Recombinant Proteins / metabolism
  • Restriction Mapping
  • Viral Proteins*

Substances

  • Peptides
  • Proteins
  • Recombinant Proteins
  • Viral Proteins
  • Cysteine Endopeptidases
  • 3C Viral Proteases