Germ line activation of the Tie2 and SMMHC promoters causes noncell-specific deletion of floxed alleles

Physiol Genomics. 2008 Sep 17;35(1):1-4. doi: 10.1152/physiolgenomics.90284.2008. Epub 2008 Jul 8.

Abstract

Tissue-specific knockouts generated through Cre-loxP recombination have become an important tool to manipulate the mouse genome. Normally, two successive rounds of breeding are performed to generate mice carrying two floxed target-gene alleles and a transgene expressing Cre-recombinase tissue-specifically. We show herein that two promoters commonly used to generate endothelium-specific (Tie2) and smooth muscle-specific [smooth muscle myosin heavy chain (Smmhc)] knockout mice exhibit activity in the female and male germ lines, respectively. This can result in the inheritance of a null allele in the second generation that is not tissue specific. Careful experimental design is required therefore to ensure that tissue-specific knockouts are indeed tissue specific and that appropriate controls are used to compare strains.

MeSH terms

  • Alleles*
  • Animals
  • Female
  • Germ Cells / metabolism*
  • Integrases / genetics
  • Integrases / metabolism
  • Male
  • Mice
  • Mice, Knockout
  • Mice, Transgenic
  • Myosin Heavy Chains / genetics*
  • Myosin Heavy Chains / metabolism
  • Promoter Regions, Genetic*
  • Receptor, TIE-2 / genetics*
  • Receptor, TIE-2 / metabolism
  • Sequence Deletion / genetics*
  • Transgenes

Substances

  • Receptor, TIE-2
  • Cre recombinase
  • Integrases
  • Myosin Heavy Chains