Infiltration of nestin-expressing cells in interstitial fibrosis in chronic cyclosporine nephropathy

Transplantation. 2008 Aug 27;86(4):571-7. doi: 10.1097/TP.0b013e3181820470.

Abstract

Background: Nestin-expressing cells play a role in the repair process of injured tissues and organs. This study examined the nestin-expressing cells in interstitial fibrosis in experimental chronic cyclosporine A (CsA) nephropathy.

Methods: Sprague Dawley rats were treated daily for 1 or 4 weeks with CsA (15 mg/kg) or vehicle (VH; olive oil, 1 mg/kg). Nestin mRNA expression was evaluated with reverse transcriptional-polymerase chain reaction, and nestin-expressing cells were detected immunohistochemically. Localization of nestin was performed with double labeling studies for vimentin, aquaporin 1, or calbindin D28K.

Results: Nestin mRNA expression was not different between VH- and CsA-treated rat kidneys. Nestin-expressing cells were rarely observed in the cortex in the VH group, but CsA-induced renal injury caused an increase in nestin-expressing cells in the cortex in a time-dependent manner. Nestin-expressing cells in the CsA group were localized to the area of interstitial fibrosis, and the number of nestin-expressing cells well correlated with the score of interstitial fibrosis (r=0.898). Nestin-expressing cells did not express vimentin, aquaporin 1, or calbindin D28K.

Conclusions: CsA-induced renal injury recruits nestin-expressing cells to injured areas, and these cells might be involved in reparative fibrosis in the progression of chronic CsA nephropathy.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cyclosporine / toxicity*
  • Cystitis, Interstitial / chemically induced
  • Cystitis, Interstitial / genetics
  • Humans
  • Immunosuppressive Agents / toxicity
  • Intermediate Filament Proteins / genetics*
  • Kidney / drug effects
  • Kidney / pathology*
  • Kidney Diseases / chemically induced
  • Kidney Diseases / genetics*
  • Kidney Diseases / pathology*
  • Nerve Tissue Proteins / genetics*
  • Nestin
  • Organ Preservation / methods
  • RNA, Messenger / genetics
  • Reverse Transcriptase Polymerase Chain Reaction

Substances

  • Immunosuppressive Agents
  • Intermediate Filament Proteins
  • NES protein, human
  • Nerve Tissue Proteins
  • Nes protein, rat
  • Nestin
  • RNA, Messenger
  • Cyclosporine